The mechanism by which OLL2712 lactic acid bacteria (1) reduce belly fat (total abdominal fat) and (2) lower the level of HbA1c (hemoglobin A1c) is thought to be that the bacteria reach the small intestine and trigger immune cells to produce an anti-inflammatory substance (IL-10), thereby suppressing the chronic inflammation in the visceral fat tissue and improving insulin resistance.
(1) The reduction in belly fat is thought to be the result of suppressing the vicious cycle in which excessive insulin secretion promotes fat synthesis, and (2) the decrease in HbA1c is thought to be the result of improved daily blood sugar control thanks to the improved insulin resistance.