Ingestion of OLL2712 lactic acid bacteria reduces abdominal fat and HbA1c concentration through the following mechanism of action, according to existing research: when the ingested OLL2712 lactic acid bacteria reaches the small intestine, it triggers immune cells to produce an anti-inflammatory substance (IL-10), which in turn suppresses the chronic inflammation in the visceral fat tissue and apparently improves insulin resistance.
The reduction in abdominal fat is thought to be a result of OLL2712 lactic acid bacteria disrupting the vicious cycle of excessive insulin secretion and fat synthesis. In addition, the decrease in HbA1c is considered to be a result of improved insulin resistance leading to better daily blood sugar regulation.